Androgenetic Alopecia vs Telogen Effluvium: same shedding, different storylines
If your shower drain looks like it’s starting a wig collection, you’re probably wondering what you’re dealing with: androgenetic alopecia vs telogen effluvium. From a distance they can look alike, but under the hood they’re very different, like a slow dimmer switch (AGA) versus a sudden power outage (TE).
As an acupuncturist and TCM practitioner with 10+ years in clinic, I look at hair loss on two layers: the dermatology pattern you can see, and the “growth systems” you feel – circulation, nutrient absorption, hormone signaling, and scalp repair. When those systems switch off, good products underperform. Turn them back on, and your routine finally starts working.
Quick definitions
- Androgenetic alopecia (AGA): genetically/hormonally driven miniaturization. Hairs get thinner and shorter over time, especially on the top/front in women and crown/hairline in men.
- Telogen effluvium (TE): diffuse shedding that kicks in weeks to months after a stressor (illness, surgery, crash dieting, postpartum, major life stress). Lots of hairs enter resting phase together, then exit dramatically.
How they act different
- Timeline: AGA is gradual and persistent; TE is abrupt, often 2–3 months after the trigger, and usually self-limited once the cause resolves.
- Look/feel: AGA thins density and part width over time; TE keeps the hairline but sheds handfuls, making the ponytail skinnier fast.
- Hope meter: TE often regrows once the trigger is handled; AGA needs ongoing support and strategy.
What the microscopes say (the evidence)
A 2022 study comparing female AGA and TE used dermoscopy (think: dermatologist’s magnifying lens) to tell them apart. Highlights you can actually use:
- AGA shows “hair diameter diversity” (>20%), meaning a mix of thick and mini hairs across the same area—classic follicle miniaturization.
- More single-hair follicular units and more vellus (baby) hairs in AGA, especially frontally versus the occiput.
- Yellow dots (empty, sebum-filled follicles) and perifollicular discoloration are more common in AGA.
- TE rarely shows HDD >20%; you might see mild HDD (>10%) and diffuse signs, but not the miniaturization signature of AGA.
Cheat sheet you can screenshot
- Cause: AGA = hormone/genetic sensitivity → miniaturization; TE = synchronized shed after stress.
- Pattern: AGA = patterned thinning (front/top); TE = diffuse shedding with preserved hairline.
- Dermoscopy: AGA = HDD >20%, ↑ single units, ↑ vellus, yellow dots; TE = HDD usually ≤10%, no miniaturization signature.
- Course: AGA is progressive without treatment; TE tends to recover once the trigger is fixed.
What to actually do
If it’s leaning AGA
- Confirm with a clinician (dermoscopy helps).
- Support the systems: improve scalp blood flow, digestion/absorption, hormone balance, and barrier health so topicals can work.
- Consider evidence-based treatments and track photos monthly.
If it’s leaning TE
- Find and fix the trigger (iron deficiency, thyroid shifts, illness, crash diets, meds, major stress).
- Replete nutrition, reduce inflammatory load, sleep like it’s your job.
- Expect a delayed but steady recovery. Shedding eases first, density returns over months.
Want a plan that turns your “growth systems” back on?
If you’re stuck between harsh meds you don’t want and “natural” fixes that never seem to stick, my 30-Day Pattern Mapping™ Reset helps restore circulation, absorption, hormone signaling, and scalp repair, so what you’re already doing finally shows up on your head.
